Thanks for your interest in the measles TWiV. If I said “just”, that was certainly a mis-statement, since immunosuppression caused by viruses is a complex, and likely multifactorial, process. However, your point is a good one: presumably loss of IL-12 would skew toward a Th2 profile, which you would think would be helpful against bacteria and parasites. But remember that T and B cells are highly specific, so it's likely that the “skewing” only affects the measles specific T cells, not all T cells. Even so, why would MV-infected people be more susceptible to bacteria and parasites? To my knowledge, we don't yet have an answer for this, but as I mentioned, this is a really active field, and a number of other strong hypotheses are being tested that might provide some clues as to how a MV infection might influence the efficiency of the entire host response.